What Makes SARS-CoV-2 More Infectious.

The Role Of The RBM:ACE2 Interface. A little over a year ago, we set out how increased infectiousness meant greater vulnerability, i.e. a new variant may be more infectious but it is also more susceptible to destruction by the innate immune response. The chances are that most people now know what a structural spike protein […]

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How Each Of Us Causes Every Variant.

The Four Nucleobases. Almost exactly twelve months ago, we explained how increased transmissibility also meant greater vulnerability, i.e. a new variant may be more infectious but it is also more susceptible to detection by the immune system. This was first evidenced by our study of SARS-CoV-2-SG614, also one year ago now. Despite there being virtually […]

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The ‘Third Wave’ That Has Been Here All Along.

B1.1.7 v B1.1.28 v B1.351. As we have previously explained, SARS-CoV-2 neither thinks nor acts geographically. Yet the misleading designation of SARS-CoV-2 variants on a geographical basis continues to dominate media coverage and government obsession, with the putative ‘third wave’ being down to either the Brazilian variant or the UK variant dependent upon whether you […]

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N501Y Is More Infectious But NOT More Dangerous.

Infectiousness Or Infectivity. If the government is to be believed, the new killer mutant of death (no, not vaccines minister Zoonotic Gaddafi, the other killer mutant of death known as N501Y) is even more infectious and even more transmissible. Except the government likes to confuse viral infectiousness with disease severity. They are very different. Also, […]

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